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Experimental and clinical findings in the last few years suggest that ferroptosis plays a role in both chronic and acute CNS diseases via disease-specific molecular mechanisms involving iron dysregulation, antioxidant deficiency, mitochondrial dysfunction and neuroinflammation

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Some key responsibilities of NAD+ include: Fuelling mitochondrial energy production which gives power to all cellular processes Supporting DNA repair mechanisms Activating longevity-associated proteins, improving cell resistance to stress and metabolism Regulating circadian rhythms and communication between cells Just like glutathione, NAD+ levels decline with stress and age